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重要的研究发现

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细胞内钾离子相对不足与健康和疾病

 

 

 

代表性论文链接 >>>>>>

 

发现单个神经细胞内模拟生物光子刺激导致生物光子在神经环路跨突触传递


Na Liu, Zhuo Wang and Jiapei Dai. Intracellular simulated biophoton stimulation and transsynaptic signal transmission. Appl. Phys. Lett. 121, 203701 (2022). https://doi.org/10.1063/5.0128956

 

研发出基于特斯拉放电弱光成像检测气溶胶中病毒的新技术 (T-Dai)


Zuxin Li，Chengming Xia，Yi Tian, Beilei Zhang，Wan Zhou，Na Liu，Qiao Wei，Jinzhong Li，Cong Chen，Linhua Chen，Zhuo Wang, Yan Sun, Chang Li，Dong Yang，Wangyang Yu，Meng Liu，Jiapei Dai. Real-time detection of viruses in aerosols with weak light imaging based on Tesla Discharge. Appl. Phys. Lett. 121, 060601 (2022); https://doi.org/10.1063/5.0104527

 

使用超弱生物光子成像系统（UBIS）发现脑缺血再灌注诱导了迟发性功能性神经回路损伤，为研究脑缺血再灌注损伤机制和新药研发提供了一种新的功能评估方法。

 

Chai W, Zhang C, Dai J. Biophoton imaging identification of delayed functional neural circuit injury after cerebral ischemia-reperfusion. J Neurosci Methods. 2022 Feb 1;367:109438. DOI:10.1016/j.jneumeth.2021.109438

 

发现了神经递质作用的量子机制，可能为阐明大脑功能和开发“量子药物”和“量子疗法”提供新思路。

 

Han Z, Chai W, Wang Z, Xiao F, Dai J. Quantum energy levels of glutamate modulate neural biophotonic signals. Photochem Photobiol Sci. 2021 Mar;20(3):343-356. DOI:10.1007/s43630-021-00022-0

 

 

发现神经递质的协同作用导致的生物光子活动和传递可能介导了意识起源和意识状态水平的转换，提出“光量子脑”新概念。

 

Chai W, Han Z, Wang Z, Li Z, Xiao F, Sun Y, Dai Y, Tang R, Dai J. Biophotonic Activity and Transmission Mediated by Mutual Actions of Neurotransmitters are Involved in the Origin and Altered States of Consciousness. Neurosci Bull. 2018 34(3):534-538. doi:10.1007/s12264-018-0215-9.

 

 

 

发现神经递质谷氨酸诱导神经细胞生物光子活动（辐射）从低等动物到人呈现光谱红移现象。这一发现对于解释人类高智力提供了新颖的科学证据。

 

  1. Wang Z, Wang N, Li Z, Xiao F, Dai J. Human high intelligence is involved in spectral redshift of biophotonic activities in the brain. Proc Natl Acad Sci U S A. 2016 Aug 2;113(31):8753-8.

 

  4. Dai J, Wang Z, Li Z, Xiao F. Reply to Salari et al.: Toward understanding the deep mechanisms regarding the biophotons related to human intelligence. Proc Natl Acad Sci U S A. 2016 Sep 20;113(38):E5542-3.

 

http://www.the-scientist.com/?articles.view/articleNo/47099/title/Do-Brighter-Species--Brains-Emit-Redder-Light-/

 

http://www.cankaoxiaoxi.com/science/20160813/1266801.shtml

 

 

 

发现了视网膜光感细胞暗噪声的起源与温度相关的生物光子活动有关，解决了长期以来理论分析和实验结果不一致的争论。

 

Li Z, Dai J. Biophotons Contribute to Retinal Dark Noise. Neurosci Bull. 2016;32(3):246-52.

 

 

 

  • 研制出了超弱生物光子成像系统（ultraweak biophoton imaging system, UBIS）和相关的分析方法。

 

  • 戴甲培，汤仁东，王超。自动化的超弱光成像系统及操作方法。2014.1.29-2033.10.30。ZL201310524951.4 (发明专利）。

 

  1. 汤仁东,戴甲培. 超弱发光成像的分水岭法光子信息提取.光电子· 激光.2013,4（1）：202-208。

 

Tang R, Dai J. Spatiotemporal imaging of glutamate-induced biophotonic activities and transmission in neural circuits. PLoS One. 2014, 15;9(1):e85643.

 

 

 

 

 

发现神经递质谷氨酸诱导神经细胞生物光子活动（辐射）的特征性改变，提出了并验证了生物光子传递（biophotonic transmission) 新机制，为量子脑机制的建立提供了新思路。

 

Tang R, Dai J. Spatiotemporal imaging of glutamate-induced biophotonic activities and transmission in neural circuits. PLoS One. 2014, 15;9(1):e85643.

 

  1. Tang R, Dai J. Biophoton signal transmission and processing in the brain. J Photochem Photobiol B. 2014,139:71-5.

 

 

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  2. 在AD脑发现了轴突漏（axonal leakage) 神经病理改变并验证了与AD风险因子（脑中风和糖尿病脑病）的相关性联系。

 

  4. Zhou Y, Luo Y, Dai J. Axonal and Dendritic Changes are Associated with Diabetic Encephalopathy in Rats: An Important Risk Factor for Alzheimer's Disease. J Alzheimers Dis.2013,34(4):937-47.

 

  12. Zhang Q, Gao T, Luo Y, Chen X, Gao G, Gao X, Zhou Y, Dai J.Transient focal cerebral ischemia/reperfusion induces early and chronic axonal changes in rats: its importance for the risk of Alzheimer’s disease. Plos One 2012, 7(3): e33722.

 

  2. Xiao A, He J, Wang Q, Luo Y, Sun Y, Zhou Y, Guan Y, Lucassen P, Dai J. The origin and development of plaques and phosphorylated tau are associated with axonopathy in Alzheimer’s disease. Neurosci Bull 2011, 27(5): 287-299.

 

研制出了“原位生物光子自显影”技术并发现了生物光子能在神经纤维上传递。

 

Sun Y, Wang C, Dai J. Biophotons as neural communication signals demonstrated by in situ biophoton autography. Photochem. Photobiol. Sci. 2010, 9:315-322.

 

 

 

 

 

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